Balance and Periodic Paralysis

Balance and Periodic Paralysis

Hello All,

I constantly discuss staying ‘totally balanced’ in all ways in order to manage our symptoms…but what does that mean exactly?? The following is my explanation.

Periodic Paralysis is a Mineral Metabolic Disorder, which is also known as an ion channelopathy. This means that the levels of the minerals/electrolytes/ions in the blood can become abnormal or can fluctuate in error depending on several issues. There will be either not enough minerals in the blood or too many minerals in the blood. Many things we call ‘triggers’ can cause this for us including, but not limited to, drugs, IV’s anesthesia, some foods (junk food, processed foods-anything not natural), food fillers and dyes, exercise, exertion, temperature fluctuations, heat, cold, stress, sleep, sodium, sugar, carbohydrates.

For those of us with varying forms of Periodic Paralysis, when a trigger is introduced in our bodies, the minerals shift and it causes many symptoms, most notably, periods of paralysis. These can be either partial or full-body. Therefore it is important for us to stay balanced.

First, this means we must keep the electrolytes/minerals in balance. These include:

potassium (K+) Use potassium meter to monitor levels.

magnesium (Mg++)

sodium (Na+)

chloride (Cl-)

calcium (Ca++)

bicarbonate (HCO3-)

phosphate (HPO4–)

Second, other things that need to be well balanced:

Sugar/glucose: Use glucose meter (blood), follow a diabetic diet and eat smaller amounts more often to prevent sugar highs and lows.

pH/acid/alkaline: Use pH strips (urine and saliva), follow a pH or alkaline balanced diet.

Hydration/dehydration: Drink plenty of water, but not too much. Low or high levels of water/fluid affect mineral levels.

.

Salt/sodium: Salt can be a trigger, use low sodium foods with care, the salt in those foods is replaced with potassium for flavor so will increase potassium levels.

Body temperature: Use a thermometer. Fever can create symptoms. Heat and cold also causes symptoms.

Vitamins: Any vitamin imbalance can create symptoms.

Exercise/exertion: Too much exertion or exercise sets our symptoms into motion, it is important to know your own limits.

Blood pressure: Monitor with a blood pressure wrist cuff.  Blood pressure needs to be kept at normal levels. This can be done with diet and staying balanced.

Oxygen levels: Monitor with finger pulse oximeters, below 95% the cells begin to be starved causing damage. If blood pressure is up or down, oxygen may be needed to avoid damage. Low oxygen levels prevent the cells from working properly and it affects the brain, heart and energy levels.

Heartbeat: Monitor with finger pulse oximeters and blood pressure wrist cuff. If heart rate is too slow the heart has to work harder and the brain and other organs are not getting the oxygen they need and if the heart is beating too fast the organs and other tissue is being deprived of oxygen.

Carbohydrates: Too many carbohydrates in a meal affect HypoKPP, but HyperKPP needs more carbohydrates in the diet.

Sleep: A lack of sleep causes chemical imbalances in the body and affects insulin levels, among other issues.

Stress (good or bad): This causes adrenaline to rise, which lowers potassium levels and also affects insulin levels.

Drugs/medications/over-the-counter: Avoid at all cost. These will cause serious imbalances in many different ways, depending on their composition and our form of Periodic Paralysis and co-existing conditions.

If we can keep our bodies in balance, we can minimize our symptoms and improve our quality of life. It is a constant battle and not easy but the results are well worth it. We can be the best we can be, naturally, by following the above ideas. We equate it to constantly walking a tightrope.

This form helps to monitor the above issues in order to stay in balance.

Until later.....

What is an Ion Channelopathy?

Hello All,

Periodic Paralysis is a channelopathy and channelopathies are considered a class four metabolic disorder. Periodic Paralysis is a fourth class mineral metabolic disorder. Today's blog is about ion channelopathies.


What is an Ion Channelopathy?

Periodic Paralysis is a rare condition like no other. It is called an ion channelopathy, which is a dysfunction of an ion channel. Ion channelopathies were first recognized in 1971 and Periodic Paralysis was one of the first to be discovered. ^{1, 2}

 Ion channels are like a microscopic tunnel in the cells of muscles. The tunnels are called muscle fibers. Ions, which are molecules or atoms, flow in and out of the muscle cells through membranes or gates. Each of the gates is shaped exactly for the correct ion or molecule to enter. The ions are made up of what we call minerals, electrolytes or proteins. Some of the common ions are potassium, sodium, magnesium, chloride and calcium. They are electrically charged and each has its own size or shape, so to speak. If the gates or membranes are faulty in size or shape, an inefficient or improper flow through the membranes can and does cause muscle weakness and paralysis because they regulate contraction and relaxation of the muscle.

Through research, I have discovered that channelopathies are classified as a class four metabolic disorder. Disorders of metabolism are usually inherited and are involved in  chemical and physical processing, which use and make energy in the body. These processes include: breathing, circulation of blood, food and nutrient digestion, elimination of waste through bowel and bladder and temperature regulation.

Unfortunately, ion channelopathies are not usually categorized nor listed in medical writing or studies as metabolic disorders. This poses a problem for recognition, diagnosis and treatment by physicians and other medical professionals. I have found and heard Periodic Paralysis, which is a channelopathy, referred to as, a neuromuscular disease (affecting muscles and/or nerves also known as myoneural), a muscular dystrophy (wasting of muscle and eventual early death), and a disease of the nervous system (nothing to do with the nervous system), therefore, none of these is correct. Periodic Paralysis is a metabolic disorder, a condition which is based in the faulty cellular level of how energy is produced in our bodies. ³

To clarify, even further, Periodic Paralysis is a channelopathy which is a mineral metabolic disorder. Metabolism disorders involving minerals are conditions in which there is either not enough or an overabundance of minerals in an individual’s blood. Minerals have many functions in metabolism and the functions of the human body. They are important in bone and muscle building and growth. Organs, cells and tissues need minerals in order to function properly. So, a dysfunction of minerals in the body affects many processes and functions. Potassium, the main mineral involved in Periodic Paralysis, is involved in making proteins from the amino acids and plays a role in carbohydrate metabolism, so a dysfunction involving it, can affect more than just muscles. ^{1, 2}

 

1. Wikipedia. (February 2013) Ion Channel. Retrieved from:  

http://en.wikipedia.org/wiki/Ion_channel

2. Wikipedia. (December 2012). Channelopathy. Retrieved from: 

http://en.wikipedia.org/wiki/Channelopathy

3. WikiDoc. (August 2012). Metabolic disorder. Retrieved from: 

http://www.wikidoc.org/index.php/Metabolic_disorder

More information:Oxford Journals. (2002). Ion Channel Diseases. Retrieved from:

 http://hmg.oxfordjournals.org/content/11/20/2435.full

Added 1/25/2017

***An Introduction to Metabolic Disorder (Mineral metabolic disorder= Channelopathy)

http://www.encognitive.com/node/1181

A fourth class, the channelopathies (some of which cause periodic paralysis and/or malignant hyperthermia) could be considered to be metabolic disorders as well, though they are not always classified as such. These disorders affect the ion channels in the cell and organelle membranes, resulting in improper or inefficient transfer of ions through the membranes.


***Mineral Metabolism Disorders
http://careers.maimonidesmed.org/Main/AdamMultimediaEncyclopedia/Mineral-metabolism-disorders-1007271.aspx

Definition

Mineral metabolism disorders are abnormal levels of minerals -- either too much or too little -- in the blood.
Minerals are very important for the human body. They have various roles in metabolism and body functions. They are essential for the proper function of cells, tissues, and organs.
Some minerals, such as iron, make up part of many proteins and enzymes in the body. Others, such as potassium, help to produce proteins from amino acids and are involved in carbohydrate metabolism. Minerals also play a role in the building of muscle and bone and are important for normal body growth.
Metabolism refers to all the physical and chemical processes in the body that create and use energy, such as:

• Breathing
• Circulating blood
• Digesting food and nutrients
• Eliminating waste through urine and feces
• Regulating temperature

Causes

Disorders of mineral metabolism are sometimes passed from parents to their children through genes. Other medical conditions, such as starvation, diarrhea, or alcoholism, can cause mineral metabolism problems.

Disorders

Minerals that play a large role in the body include:

• Calcium
• Magnesium
• Phosphorus
• Potassium
• Selenium
• Sodium

Disorders in which mineral metabolism problems often occur include:

• Disorders of phosphorus metabolism:
• Hypophosphatemia
• Osteomalacia
• Rickets
• Rhabdomyolysis
• Hyperparathyroidism
• Hypoparathyroidism
• Disorders of potassium metabolism:
• Bartter syndrome
• Periodic paralysis with hypokalemia***
• Hypokalemic periodic paralysis***
• Hyperaldosteronism - primary and secondary
• Cushing’s disease
• Proximal renal tubular acidosis
• Distal renal tubular acidosis
• Fanconi’s syndrome
• Addison’s disease
• Kidney disease
• Disorders of iron metabolism:
• Hemochromatosis
• Cirrhosis
• Disorders of copper metabolism:
• Wilson’s disease
• Menkes syndrome
• Disorders of calcium metabolism:
• Hyperparathyroidism
• Nephrocalcinosis
• Pseudohypoparathyroidism
• Hypercalcemia
• Osteoporosis
• Movement - unpredictable or jerky
• Kidney stones
• Milk-alkali syndrome
• Paget’s disease
• Multiple endocrine neoplasia (MEN 1)
• Osteomalacia
• Rickets
• Disorders of sodium metabolism:
• Dilutional hyponatremia (SIADH)
• Hypernatremia
• Disorders of magnesium metabolism:
• Hypomagnesemia
• Hypermagnesemia
• Disorders of selenium metabolism
• Selenium deficiency
• Selenium excess
  
  >>>>>>>>>>>>>>>>>>>>>>

Other Disorders Involving Metabolism Of Minerals











Public Health Service Publication No. 1693
https://books.google.com/books?id=icIaAAAAMAAJ&pg=PA153&lpg=PA153&dq=mineral+metabolism++disorders+periodic+paralysis&source=bl&ots=b-rjeyEFyj&sig=Dy_C8ZLRQGtzoqLoBRaxW1p9qQI&hl=en&sa=X&ved=0ahUKEwiY0Zy7j97RAhUQzWMKHbxFBDEQ6AEIUTAJ#v=onepage&q=mineral%20metabolism%20%20disorders%20periodic%20paralysis&f=false

Metabolic Disorder
http://www.wikidoc.org/index.php/Metabolic_disorder

Ion Channels list
http://www.nature.com/ncb/journal/v6/n11/fig_tab/ncb1104-1039_T1.html

PP IS A MINERAL METABOLIC DISORDER http://healthmedicinet.com/ency/article/007271.htm
Mineral metabolism disorders - Health Medicine Network healthmedicinet.com/ency/article/007271.htm

Until later...

What is Andersen-Tawil Syndrome?

Hello All,

I have been diagnosed with the rarest form of Periodic Paralysis called  Andersen-Tawil Syndrome. I was first diagnosed based on my symptoms and characteristics. This is called a "clinical diagnosis."  I now have a genetic diagnosis (KCNJ5). Andersen-Tawil Syndrome (ATS), given a name in 1971 was the first ion channelopathy discovered. It is a very rare form of Periodic Paralysis, a mineral metabolic disorder. ATS is an autosomal dominant disorder, which means it is usually passed by one parent to the child. If someone is a carrier his or her children have a 50% chance of being born with it or as a carrier. ATS accounts for approximately 10% of all periodic paralysis cases. It is characterized by three particular components: periods of paralysis from high, low or normal potassium levels, distinctive craniofacial and skeletal characteristics and long QT interval heartbeat with a predisposition toward life-threatening ventricular arrhythmia. However, affected individuals may express only one or two of the three components and they may be very subtle.

Some of these components and symptoms were found in my mother, possibly through her father, and many of her descendants; specifically, two of her three sons and me, her only daughter. Many of her grandchildren and great-grandchildren also have varying degrees of the characteristics and symptoms.

Some of the manifestations of this condition are serious and life-threatening. Each family member of someone diagnosed with ATS should be well educated about this syndrome, in particular, regarding the episodes of paralysis (full body or partial), the heart complications, the strange effects of most medications and the serious complications of anesthesia.

An individual is born with Andersen-Tawil Syndrome and symptoms may begin in early childhood or not until later in life. Members of the same family can have varying degrees of it or some of the characteristics without actually having it.

During an attack, brought on by many triggers to include: carbohydrates, sugar, medications, exercise, heat, cold, periods of sitting too long, stress (good or bad), etc; potassium leaves the organs it belongs in and goes into the muscles where it does not belong and paralyzes the muscles (totally or partially). The depletion of the potassium in the other organs can cause symptoms such as irregular heartbeat, weakness, fainting, numbness, tingling, breathing issues, choking/swallowing problems, exercise intolerance, etc. After many years the body can become permanently weakened.

It is important to get a diagnosis for proper treatment to avoid the permanent disabilities. It is also important so one can avoid the triggers and hopefully control the episodes as much as possible. Most over-the-counter medications, as well as those prescribed by physicians, such as antibiotics and painkillers, can cause serious consequences.  Many medications can cause an opposite effect such as sleeping aids can keep you awake and agitated.

Executive Functioning (EF) Disorder can accompany ATS. There are three primary layers of executive functions: self-regulation, organization and high order reasoning skills. It is associated with many disabilities: Attention Deficit Hyperactivity Disorder (AD/HD), Learning Disabilities (LD), Tourette Syndrome (TS), Obsessive-Compulsive Disorder (OCD), Autism, Depression, Bipolar, etc. It is important to have these conditions diagnosed as early as possible for proper treatment, training, and education.

To better explain and describe ATS I am including the following information I have collected over my years of research for the "Characteristics of Andersen-Tawil Syndrome." There are more than the 5 or 6 we typically read. Several articles are sighted for this section due to the differences in symptoms in some of them. Some personal information has been added for clarity.

Symptoms & Characteristics

Several articles are sighted in the following section due to the differences in symptoms in some of them. Some personal information has been added for clarity.

Anderson-Tawil syndrome is a disorder that causes episodes of muscle weakness (periodic paralysis), changes in heart rhythm (arrhythmia), and developmental abnormalities. The most common changes affecting the heart are ventricular arrhythmia, which is a disruption in the rhythm of the heart's lower chambers, and long QT syndrome. Long QT syndrome, is a cardiac channelopathy, which causes the heart (cardiac) muscle to take longer than usual to recharge between beats. If untreated, the irregular heartbeats can lead to discomfort, fainting (syncope), or cardiac arrest.

 

At this time, there are two recognized types of Andersen-Tawil syndrome, Type 1 and Type 2, which are distinguished only by their genetic causes. Type 1 has a known genetic mutation (either one of two forms KCNJ2 and KCNJ5). The other, Type 2, though the symptoms and characteristics are exactly the same, has no known genetic mutation, which has been discovered yet.  

Physical abnormalities associated with Andersen-Tawil syndrome typically affect the head, face, and limbs. These features often include a very small lower jaw (micrognathia), dental abnormalities, low-set ears, widely spaced eyes, and unusual curving of the fingers or toes (clinodactyly). Some affected people also have short stature and an abnormal curvature of the spine (scoliosis).

 

Symptoms

·Widely spaced eyes
· Short stature
· Scoliosis
· Webbed toes or fingers
· Unusual short fingers
· Low set ears
· Broad forehead
· Small jaw
· Protruding jaw
· Broad nasal root
http://en.wikipedia.org/wiki/Andersen–Tawil_syndrome

Disease characteristics.

Andersen-Tawil syndrome (referred to as ATS in this entry) is characterized by a triad of episodic flaccid muscle weakness (i.e., periodic paralysis), ventricular arrhythmias and prolonged QT interval, and anomalies such as low-set ears, ocular hypertelorism, small mandible, fifth-digit clinodactyly, syndactyly, short stature, and scoliosis. Affected individuals present in the first or second decade with either cardiac symptoms (palpitations and/or syncope) or weakness that occurs spontaneously following prolonged rest or following rest after exertion. Mild permanent weakness is common. Mild learning difficulties and a distinct neurocognitive phenotype (i.e., deficits in executive function and abstract reasoning) have been described.
www.ncbi.nlm.nih.gov/pubmed/20301441

Executive Functioning (EF) Disorder

Relates to difficulty in self regulation, organizing, integration, or high order reasoning skills.

"Executive Function disorder, is a disability of not being able to show what you know"

Executive Function disorder is associated with many disabilities: Attention Deficit Hyperactivity Disorder (AD/HD), Learning Disabilities (LD), Tourette Syndrome (TS) , Obsessive Compulsive Disorder (OCD), Autism, Depression, Bipolar, etc.

Most people with AD/HD also have Executive Function Disorder, but someone can have Executive Function Disorder without being diagnosed with a disability. Executive Function program and services would be needed if the student progress in the general growth in the acquisition of knowledge and skills are being negatively impacted.

Eight Pillars of Executive Control

 by Dr. Adam J. Cox, Ph.D., January 2009,

Minuteman Technical High School SEPAC presentation

1. Initiating Action being able to organize one’s thoughts well enough to get started on a particular task without having to be asked multiple times.
2. Flexible Thinking involves learning to adapt by shifting one’s focus and pace as various situations unfold. Imagine how difficult it would be to drive your car if it wouldn’t turn and only went one speed. (About as difficult as parenting a child with only one speed and one direction!)
3. Sustaining Attention focusing long enough and accurately enough to learn important information. By extension, attention also involves the ability to block distraction. A well-orchestrated “executive brain” knows its priorities.
4. Organization is about managing space. It’s also about taking the emotional impact of chaos seriously. Why? Because chronic disorganization undermines forward momentum – a sense of accomplishment.
5. Planning is about managing time, and is more important than any other executive pillar when it comes to finishing things on schedule. A planning mind uses time as a tool to clarify priorities and enhance productivity; indispensable skills to 21st century success, beginning with school and, eventually, careers.
6. Working Memory is the ability to retain information long enough for it to be stored in long-term memory. Our society has a word for this process – learning. Of all the executive controls, working memory is the most pervasive, contributing to the smooth operation of every pillar. (Working memory is the rocket fuel of the modern mind.)
7. Self-Awareness pertains to having both sufficient self-knowledge and an ability to perceive how others see you. This information is essential to making purposeful choices about how to act in situations where one wants to avoid unintended consequences that lead to isolation or ostracism.
8. Regulating Emotions means expressing one’s feelings in proportion to the events that elicited them. When a child under or over-reacts, she is out-of-sync with people or particular events. Socially, people tend to ignore a silent recluse, and run away from an “erupting volcano.”
From his book: No Mind Left Behind: Understanding and Fostering Executive Control- The Eight Essential Brain Skills every Child Needs to Thrive, by Adam J. Cox, Ph.D. Psychologist
Executive Function Skills, by Sarah Ward, MS, CCC-SLP, February, 2007 SPED PAC Presentation

3 Primary Layers of Executive Functions:

1.      Self-Regulation

a.      Awareness

b.      Motivation

c.       Initiation

d.      Emotional Control

2.      Self-awareness

a.     Self monitor the ability to inhibit or delay responding, which permits impulse control resistance to distraction and delay of gratification)

b.      Metacognition (Learning how to learn)

3.      Organization and Integration

a.      Integrate details into a bigger picture

b.     Organize and store information so it can be traced back and retrieved over time (Episodic Memory), (used for planning for the future)

4.      Higher Order Reasoning Skills

a.     Analyze

b.     Draw a conclusion

c.      Solve a problem

d.     Predict an outcome

e.     Reason

f.        Evaluate
http://concordspedpac.org/ExecutiveFunctions.html

Symptoms of Andersen-Tawil Syndrome

 

· Ventricular arrhythmia
· Abnormal heart rhythm
· Long QT syndrome increased time needed for heart to recharge after each heart beat
· Irregular heartbeat
· Discomfort
· Fainting caused by irregular heart beat
· Small lower jaw
· Dental abnormalities
· Low-set ears
· Widely spaced eyes
· Abnormal curving of fingers
· Abnormal curving of toes
· Short stature
· Abnormal curvature of the spine
http://www.wrongdiagnosis.com/a/andersen_tawil_syndrome/symptoms.htm

Distinctive physical features recognized initially included low-set ears, ocular hypertelorism, small mandible, fifth-digit clinodactyly, syndactyly, short stature, broad nasal root, and scoliosis [Andersen et al 1971, Tristani-Firouzi et al 2002, Donaldson et al 2003]. Dental enamel discoloration was noted in two kindreds with the Gly300Asp and Arg218Trp mutations [Davies et al 2005].
Detailed, prospectively collected data in ten individuals with confirmed KCNJ2 mutations have expanded the phenotype to include a characteristic facies and dental and skeletal anomalies [Yoon et al 2006a].
· Characteristic facies include broad forehead, short palpebral fissures, full nasal bridge with bulbous tip, hypoplasia of maxilla and mandible, thin upper lip, and a triangular shape.
· Dental findings include (among others) persistent primary dentition, multiple missing teeth (oligodontia), and dental crowding.
· Skeletal findings include mild syndactyly of toes 2 and 3 as well as fifth-digit clinodactyly.
· Novel findings include small hands and feet (<10th centile for age) and joint laxity.
Isolated reports of renal anomalies include unilateral hypoplastic kidney [Andelfinger et al 2002] and renal tubular defect [Davies et al 2005].
http://www.ncbi.nlm.nih.gov/books/NBK1264/

Dysmorphic Features:

May be very subtle, partial or seen in 'unaffected' family members
Skeletal:
· short stature (often);
· scoliosis

Hands/Feet:
· tapering fingers,
· clinodactyly (inward curvature/ 5th fingers);
· brachydactyly (unusually short fingers);
· syndactyly (webbing between fingers or between 2nd and 3rd toes)

 Facial:
· hypertelorism (widely spaced eyes);
· mandibular hypoplasia (small jaw);
· low-set ears;
· broad forehead;
· malar hypoplasia; (underdevelopment of cheekbones)
· broad nasal root;
· micrognathia (short jaw);
· prognathism (protruding jaw);
· ptosis; (an abnormally low position (drooping) of the upper eyelid)
· cleft palate, high arched palate

Definition of Terms for Symptoms:

· characteristic facies (characteristic appearance of the face in association with a disease or abnormality)
· brachydactyly (unusually short fingers)
· brachydactyly type D (clubbed thumbs) (characterized by a slightly shorter thumb that is round in section and larger at the end)
· clinodactyly (inward curvature/ 5th fingers);
· syndactyly (webbing between fingers or between 2nd and 3rd toes)
· small mandible (lower jaw in which the lower teeth reside and chin)
· hypoplasia of maxilla (small upper jaw)
· short palpebral fissures (short opening for the eyes between the eyelids)
· persistent primary dentition (still have some baby teeth)
· ocular hypertelorism (widely spaced eyes);
· scoliosis (curved spine)
· microcephaly (abnormal smallness of the head)
· delayed bone age (slowed degree of maturation of child's bones)
· joint laxity (looseness of the muscles and soft tissue surrounding a joint)
· broad nasal root (wide space between the inner corners of eyes)
· broad forehead (increased distance between the two sides of the forehead or top to bottom of forehead)
· malar hypoplasia (small cheekbones)
· micrognathia (short jaw);
· prognathism (protruding jaw)
· ptosis (an abnormally low position (drooping) of the upper eyelid)
· cleft palate (a congenital fissure in the roof of the mouth)
· high arched palate (roof of the mouth is high)

Facial Dysmorphology

Drawings are used to depict terminology and to illustrate certain aspects of facial variation.
http://www.peds.ufl.edu/divisions/genetics/teaching/facial_dysmorphology.htm

In most of the textbook or journal articles, Andersen-Tawil Syndrome is described as being so rare that only 100 cases have been diagnosed worldwide. This is incorrect. In a part of France alone, over 90 cases exist. 100 cases of ATS is a gross underestimation. Far more cases exist in varying forms.

 

Until later...

Periodic Paralysis and Balance November 28, 2013

Happy Thanksgiving to our readers in the USA!!!

About two weeks ago, physically, I could not even get out of my recliner and I was in a brain fog, and more. I felt miserable. I mentioned it in some of my previous blogs. I have had varying degrees of this for many, long months and it was getting worse.

I had to evaluate what the trigger was so I could stop it or avoid it. I just could not face one more day of feeling like I was dying. I studied my diet, which is organic and free of additives, pesticides and pH balanced. I monitored my vitals and my potassium and glucose levels, which were normal. I decided the only other thing out of the ordinary was my supplements. I had been taking them for nearly three years. They are pure and free of gluten, sugar, etc with no fillers. I decided to experiment, however, and stopped taking them.

Since that day...I have been able to get out of my recliner and the fog has lifted!!! My world has color again!! No more shades of gray!! I have more energy. Calvin has been shocked at the difference and continues to caution me to not do too much.

I have been able to prepare an entire Thanksgiving dinner!! Each day I made one thing and froze it or packaged it until today. I will be putting my own turkey in the oven in a little while! My daughter and son-in-law arrived last night and I was able to serve them gluten free and low sugar pumpkin pie that I made, as well as, to be able to visit with them without going into paralysis.

I believe that the problem was taking supplements that I did not need any longer. It was throwing my body out of balance. I may need them again in the future, but not now. As my symptoms wax and wane in the future, I will need to evaluate why, but I know now that I should not put anything in my body I do not need. Those of us with Periodic Paralysis must walk that fine line...We must “walk the tightrope”

Periodic Paralysis is an ion channelopathy. Ion channelopathies are mineral metabolic disorders. Periodic Paralysis is a 4^th class mineral metabolic disorder. As such, it appears that anything may affect the balance…we must continually evaluate and re-evaluate each thing we are putting into out body.

Until later…